Electrocardiography (ECG/EKG)
Definition
Electrocardiography (ECG) is the
graphic recording of the electrical activity of the heart during depolarization
and repolarization using electrodes placed on the body surface. The recorded
tracing is called an electrocardiogram (ECG or EKG).
Historical Background
Invented by Willem Einthoven, Introduced the string
galvanometer, Developed the standard limb leads (I, II, III), Nobel Prize in
Physiology/Medicine: 1924
Importance of ECG
ECG is:
- Non-invasive
- Rapid
- Inexpensive
- Highly
informative
Clinical Uses
- Diagnosis
of arrhythmias
- Myocardial
infarction (MI)
- Ischemic
heart disease
- Conduction
abnormalities
- Electrolyte
disturbances
- Chamber
hypertrophy
- Drug
toxicity
- Pericarditis
- Monitoring
ICU patients
- Pre-operative
assessment
Physiological Basis of ECG
The heart generates electrical impulses due to:
- Depolarization
- Repolarization
These currents spread through body tissues and are
detected by surface electrodes.
Cardiac Conduction System
Components
- SA
node
- Atrial
pathways
- AV
node
- Bundle
of His
- Right
and left bundle branches
- Purkinje
fibers
Sequence of Activation
SA node → atria → AV node → Bundle of His → bundle
branches → Purkinje fibers → ventricles
Principle of ECG Recording
When an electrical impulse:
- Moves
towards a positive electrode → upward deflection
- Moves
away from positive electrode → downward deflection
- Moves
perpendicular → biphasic wave
ECG Paper
Standard ECG Paper
- Speed
= 25 mm/sec
- 1
small square = 1 mm
- 1
large square = 5 mm
Time Calibration
|
Measurement |
Value |
|
1
small square |
0.04
sec |
|
1
large square |
0.20
sec |
|
5
large squares |
1 sec |
Voltage Calibration
|
Measurement |
Voltage |
|
10 mm |
1 mV |
|
1 mm |
0.1
mV |
Leads of ECG
Definition of Lead
A lead is a particular view of the heart’s electrical
activity.
Types of ECG Leads
1. Bipolar Limb Leads (Standard Leads)
Introduced by Einthoven.
|
Lead |
Connection |
|
Lead
I |
Right
arm (−) → Left arm (+) |
|
Lead
II |
Right
arm (−) → Left leg (+) |
|
Lead
III |
Left
arm (−) → Left leg (+) |
Einthoven’s Triangle
The three limb leads form an imaginary triangle around
the heart.
Lead II=Lead I+ Lead III
2. Unipolar Limb Leads (Augmented Leads)
|
Lead |
Area
Viewed |
|
aVR |
Right
side of heart |
|
aVL |
Left
upper heart |
|
aVF |
Inferior
wall |
3. Chest (Precordial) Leads
|
Lead |
Position |
|
V1 |
4th
ICS right sternal border |
|
V2 |
4th
ICS left sternal border |
|
V3 |
Between
V2 and V4 |
|
V4 |
5th
ICS mid-clavicular line |
|
V5 |
Left
anterior axillary line |
|
V6 |
Left
mid-axillary line |
Areas Viewed by Chest Leads
|
Leads |
Region
Seen |
|
V1–V2 |
Septal |
|
V3–V4 |
Anterior |
|
V5–V6 |
Lateral |
Components of Normal ECG
Main Components
- P
wave
- PR
interval
- QRS
complex
- ST
segment
- T
wave
- QT
interval
- U
wave
P Wave
Represents: Atrial depolarization
Normal Features
|
Parameter |
Normal
Value |
|
Duration |
<
0.12 sec |
|
Height |
<
2.5 mm |
Normal Characteristics
- Upright
in I, II, aVF
- Inverted
in aVR
- Biphasic
in V1
Abnormal P Waves
P Mitrale
- Broad,
notched P wave
- Left
atrial enlargement
P Pulmonale
- Tall
peaked P wave
- Right
atrial enlargement
PR Interval
Represents: Time from atrial depolarization to
ventricular depolarization
Includes:
- P
wave
- PR
segment
Normal PR Interval
PR interval=0.12 to 0.20 sec
Significance
Prolonged PR Interval
Seen in: First degree AV block
Short PR Interval
Seen in: Wolff-Parkinson-White syndrome (WPW)
QRS Complex
Represents: Ventricular depolarization
Components
- Q
wave = first negative deflection
- R
wave = first positive deflection
- S
wave = negative deflection after R
Normal QRS Duration
QRS duration<0.12 sec
Normal Duration
0.06–0.10 sec
Significance of Wide QRS
Occurs in:
- Bundle
branch block
- Ventricular
tachycardia
- Hyperkalemia
Pathological Q Waves
Suggest: Myocardial infarction
Criteria
- Width
> 0.04 sec
- Depth
> 25% of R wave
ST Segment
Represents: Early ventricular repolarization
Normally:
- Isoelectric
ST Elevation
Seen in:
- Acute
myocardial infarction
- Pericarditis
ST Depression
Seen in:
- Myocardial
ischemia
- Digoxin
effect
T Wave
Represents: Ventricular repolarization
Normal Features
- Upright
in most leads
- Inverted
in aVR
Tall T Waves
Seen in:
- Hyperkalemia
Inverted T Waves
Seen in:
- Ischemia
- Ventricular
hypertrophy
QT Interval
Represents: Total ventricular electrical activity
Measured from: Beginning of QRS → end of T wave
Normal QT Interval
QTc < 440/ ms
Prolonged QT
May cause:
- Torsades
de pointes
Causes:
- Hypocalcemia
- Drugs
- Congenital
syndromes
U Wave
Small wave after T wave.
Seen in:
- Hypokalemia
- Bradycardia
Normal ECG Values
|
Parameter |
Normal
Value |
|
Heart
rate |
60–100/min |
|
P
wave duration |
<0.12
sec |
|
PR
interval |
0.12–0.20
sec |
|
QRS
duration |
<0.12
sec |
|
QT
interval |
<0.44
sec |
|
ST
segment |
Isoelectric |
Determination of Heart Rate
Method 1: Large Box Method
Example
If RR interval = 4 large boxes
Method 2: Small Box Method
Definition
Average direction of ventricular depolarization.
Normal Axis: -30° to +90°
Axis Deviation
|
Type |
Causes |
|
Left
axis deviation |
LVH,
LBBB |
|
Right
axis deviation |
RVH,
pulmonary disease |
Rhythm Analysis
Normal Sinus Rhythm Criteria
- HR
= 60–100/min
- Regular
rhythm
- Each
P followed by QRS
- Normal
PR interval
Systematic ECG Interpretation
Stepwise Approach
Step 1: Check Calibration
- Paper
speed
- Voltage
Step 2: Determine Heart Rate
Step 3: Determine Rhythm
Step 4: Examine P Waves
Step 5: Measure PR Interval
Step 6: Assess QRS Complex
Step 7: Assess ST Segment
Step 8: Examine T Waves
Step 9: Measure QT Interval
Step 10: Determine Axis
ECG Changes in Common Conditions
1. Sinus Bradycardia
ECG Features
- HR
< 60/min
- Normal
rhythm
Causes
- Athletes
- Hypothyroidism
- Increased
vagal tone
2. Sinus Tachycardia
ECG Features
- HR
>100/min
Causes
- Fever
- Shock
- Hyperthyroidism
ECG Features
- No
P waves
- Irregularly
irregular rhythm
4. Atrial Flutter
ECG Features
- Saw-tooth
flutter waves
5. Ventricular Tachycardia
ECG Features
- Wide
QRS complexes
- Rapid
ventricular rhythm
ECG Features
- Chaotic
irregular waves
- No
organized complexes
Medical emergency.
7. Myocardial Infarction
ECG Changes
Early: Hyperacute T waves
Acute: ST elevation
Later: Pathological Q waves
Chronic: T wave inversion
Localization of MI
|
Leads |
Region |
|
II,
III, aVF |
Inferior
wall |
|
V1–V4 |
Anterior
wall |
|
I,
aVL, V5, V6 |
Lateral
wall |
Bundle Branch Blocks
Right Bundle Branch Block (RBBB)
ECG Features
- Wide
QRS
- rSR′
pattern in V1
Left Bundle Branch Block (LBBB)
ECG Features
- Broad
notched R waves in V5/V6
Electrolyte Abnormalities on ECG
|
Electrolyte
Disorder |
ECG
Finding |
|
Hyperkalemia |
Tall
T waves |
|
Hypokalemia |
U
waves |
|
Hypercalcemia |
Short
QT |
|
Hypocalcemia |
Long
QT |
Clinical Significance of ECG
ECG helps in:
- Emergency
diagnosis
- Monitoring
therapy
- Detecting
silent ischemia
- Assessing
pacemakers
- Evaluating
electrolyte imbalance
Advantages of ECG
- Simple
- Cheap
- Bedside
procedure
- Rapid
diagnosis
Limitations of ECG
- May
be normal despite disease
- Interpretation
requires expertise
- Transient
abnormalities may be missed
High-Yield Points
- P
wave = atrial depolarization
- QRS
= ventricular depolarization
- T
wave = ventricular repolarization
- PR
interval normal = 0.12–0.20 sec
- QRS
duration <0.12 sec
- ST
elevation = acute MI
- Hyperkalemia
= tall T waves
- Hypokalemia
= U waves
- Atrial
fibrillation = absent P waves
- ECG
paper speed = 25 mm/sec
Summary
Electrocardiography is one of the most essential
diagnostic tools in medicine. Understanding ECG fundamentals—including waves,
intervals, leads, axis, and interpretation—is critical for MBBS and NEET
students. Mastery of normal ECG patterns forms the basis for diagnosing
arrhythmias, myocardial infarction, electrolyte disturbances, and conduction
defects.